Microglia aging advances through intermediate states that drive inflammatory activation and cognitive decline

Direct links to NCBI, no account and no request form: the whole study as GSE211340_RAW.tar, processed values as the series matrix, the supplementary file directory, and per-sample supplementary files for any of the 6 samples. Raw sequencing reads are also available from ENA.

Also filed as BioProject PRJNA869921. Searching any of these in the dataset finder brings you back here.

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• GSE314817 Multi-tissue, multi-time-point transcriptomic atlas of aging in mice and rats [mouse] 2432 samples
• GSE331176 Phagosome-mediated activation of STING by purine and pyrimidine-based bacterial cyclic dinucleotides 380 samples
• GSE261492 Hypertension Promotes Neuroinflammation, Brain Injury and Cognitive Impairment 48 samples
• GSE302792 SenCat: Cataloging human cell senescence through multiomic profiling of multiple senescent primary cell types 33 samples
• GSE343043 Disease context dictates the cellular targets of IL-17 in inflammatory skin disease 29 samples
• GSE341985 Diseaase context dictates the cellular targets of IL-17 in inflammatory skin disease [2] 27 samples
• GSE285471 APOE genotype modifies the gene signature of microglial and astrocytic responses to aging 24 samples
• GSE338763 Inflammatory Neuropathy in Mouse and Primate Models of Colorectal Cancer 24 samples

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