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Nuclear matrix associated protein SMAR1 attenuated acute graft-versus-host disease by targeting JAK-STAT signaling in CD4+ T cells [ATAC-Seq]

GSE229932 Homo sapiens Genome binding/occupancy profiling by high throughput sequencing 6 samples Submitted 2025/04/06 Platform GPL24676
Summary
As a chromatin remodeling factor, SMAR1 might promote local chromatin changes, leading to the activation or inhibition of specific gene subsets in T cells. We performed high-resolution ATAC-seq on primary CD4+ T cells with SMAR1 overexpression (OE) and control (Ctrl), conducting a genome-wide analysis to map the locations and profile the accessibility of diverse regulatory elements.
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Also filed as BioProject PRJNA956830 and SRA study SRP433163. Searching any of these in the dataset finder brings you back here.

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