SETD2 Mutations Do Not Contribute to Clonal Fitness in Response to Chemotherapy in Childhood B Acute Lymphoblastic Leukemia (WES)
human · 12 samples
- Organism
- Homo sapiens (human)
- Samples
- 12 samples: 10 distinct titles, 1 of them repeatedGroups are read from the sample names, not from the paper. Check the list below.
- Data you can get
- A processed table (series matrix) and supplementary files from NCBI.
- Paper
- SETD2 mutations do not contribute to clonal fitness in response to chemotherapy in childhood B cell acute lymphoblastic leukemiaLeukemia & lymphoma · 2024
Direct links to NCBI, no account and no request form. Pick by what you want to do: take the small processed table to look, the supplementary files to re-analyse, or the original raw data to start over.
Processed table
The series matrix: one table of values per sample as the submitters processed it. The smallest download, and it opens in Excel or R. Start here to see what the data look like.
Supplementary files
The count tables and other files the authors uploaded. Take only the samples you need.
Whole study · RAW.tar
Everything the authors deposited as one archive (GSE231723_RAW.tar), including the raw array or instrument files. Can be large; the picker in step 2 takes just the samples you need.
Also filed as BioProject PRJNA967354. Searching any of these in the dataset finder brings you back here.
How we read this: Read from 12 sample titles: 10 distinct titles with little repetition. These groups are guessed from the sample names, not taken from the paper, so check them against the sample list below before relying on them.
- GSM7299651 KOPN cells, sgRNA empty vector control, single cell clone F9
- GSM7299652 KOPN cells, sgRNA empty vector control, single cell clone B10
- GSM7299653 KOPN cells, sgRNA empty vector control, single cell clone H12
- GSM7299654 KOPN cells, sgRNA empty vector control, single cell clone F9 after treatment
- GSM7299655 KOPN cells, sgRNA empty vector control, single cell clone B10 after treatment
- GSM7299656 KOPN cells, sgRNA empty vector control, single cell clone H12 after treatment
- GSM7299657 KOPN cells, sgRNA SETD2 clone BA, single cell clone A1
- GSM7299658 KOPN cells, sgRNA SETD2 clone BA, single cell clone A3
- GSM7299659 KOPN cells, sgRNA SETD2 clone BA, single cell clone A6
- GSM7299660 KOPN cells, sgRNA SETD2 clone BA, single cell clone A1 after treatment
- GSM7299661 KOPN cells, sgRNA SETD2 clone BA, single cell clone A3 after treatment
- GSM7299662 KOPN cells, sgRNA SETD2 clone BA, single cell clone A6 after treatment
- GSE231724 SETD2 Mutations Do Not Contribute to Clonal Fitness in Response to Chemotherapy in Childhood B Acute Lymphoblastic Leukemia 32 samples shares setd2, lymphoblastic, chemotherapy, contribute, mutations, childhood, leukemia
- GSE245139 Long circulating RNAs identified as biomarker candidates for risk stratification in childhood acute lymphoblastic leukemia 45 samples shares lymphoblastic, childhood, leukemia
- GSE243298 3D Chromatin Remodeling in B-cell Development and B-cell Acute Lymphoblastic Leukemia [Hi-C] 35 samples shares lymphoblastic, leukemia
- GSE279521 Focal deletions of a promoter tether activate the IRX3 oncogene in T-cell acute lymphoblastic leukemia [UMI4C] 13 samples shares lymphoblastic, leukemia
- GSE33315 Discovery of novel recurrent mutations and rearrangements in early T-cell precursor acute lymphoblastic leukaemia by whole genome sequencing 575 samples shares lymphoblastic, mutations
- GSE12995 Expression data for diagnosis acute lymphoblastic leukemia samples 175 samples shares lymphoblastic, leukemia
- GSE243299 3D Chromatin Remodeling in B-cell Development and B-cell Acute Lymphoblastic Leukemia 112 samples shares lymphoblastic, leukemia
- GSE272023 CpG island methylator phenotype classification improves risk assessment in pediatric T-cell Acute Lymphoblastic Leukemia [RNA-seq] 111 samples shares lymphoblastic, leukemia
Metadata from NCBI GEO, cached and refreshed periodically — the NCBI page above is authoritative. Downloads link straight to NCBI/ENA; nothing is proxied through BioTransfer.