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ChIP-seq in SCLC transformation

GSE242305 Homo sapiens Genome binding/occupancy profiling by high throughput sequencing 4 samples Submitted 2024/06/12 Platform GPL24676
Summary
SCLC transformation is one of the mechanisms of TKI resistance in lung cancer. To clarify its epigenetic changes, we used Chip-seq to detect the changes of H3K9me2 in pre-LUAD and post-SCLC transformation cells.
Published in
EHMT2-mediated transcriptional reprogramming drives neuroendocrine transformation in non-small cell lung cancer
Yang C, Ma S, Zhang J et al. · Proceedings of the National Academy of Sciences of the United States of America 2024 · PMID 38814866 · doi:10.1073/pnas.2317790121
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Also filed as BioProject PRJNA1013071 and SRA study SRP458594. Searching any of these in the dataset finder brings you back here.

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