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METTL3 Facilitates Hypertrophic Scar Fibrosis by Promoting pri-miRNA-31 Maturation and ZBTB20 Silencing in m6A-dependent Manner [RNA-Seq]

GSE246977 Homo sapiens Expression profiling by high throughput sequencing 4 samples Submitted 2024/08/30 Platform GPL16791
Summary
To clarify the role of METTL3-mediated m6A modification in hypertrophic scars (HS), we conditionally knocked out the m6A methyltransferase METTL3 in HS-derived dermal fibroblasts (HSFBs). We then performed RNA-seq analysis on normal control and Mettl3 knockdown HSFBs.
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Also filed as BioProject PRJNA1035478 and SRA study SRP470100. Searching any of these in the dataset finder brings you back here.

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