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METTL16 deficiency attenuates apoptosis through translational control of extrinsic death receptor during nutrient deprivation

GSE249176 Homo sapiens Expression profiling by high throughput sequencing; Other 6 samples Submitted 2024/03/01 Platform GPL20795
Summary
METTL16 is a well-characterized m6A methyltransferase that has been reported to contribute to tumorigenesis in various types of cancer. However, the effect of METTL16 on tumor progression under restricted nutrient conditions, which commonly occur in tumor microenvironment, has yet to be elucidated. Herein, our study initially reported the inhibitory effect of METTL16 depletion on apoptosis under amino acid starvation conditions.
Published in
METTL16 deficiency attenuates apoptosis through translational control of extrinsic death receptor during nutrient deprivation
Li Q, Yang L, Zhang C et al. · Biochemical and biophysical research communications 2024 · PMID 38520913 · doi:10.1016/j.bbrc.2024.149802
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Also filed as BioProject PRJNA1047606 and SRA study SRP475364. Searching any of these in the dataset finder brings you back here.

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