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Up-regulation of cholesterol synthesis by lysosomal defects requires a functional mitochondrial respiratory chain

GSE256471 Homo sapiens Expression profiling by high throughput sequencing 10 samples 2024/05/24 GPL15433
Summary
To understand the common and opposite effects of mitochondrial and lysosomal perturbations on cellular signaling, we performed RNAseq in HeLa cells stably silenced for the mitochondrial respiratory chain subunit UQCRC1 (as a model of chronic mitochondrial respiratory chain deficiency), for lysosomal hydrolase acid alpla-glucosidase (GAA), or for lysosomal cathepsin B (CTSB). The controls were HeLa cells with scrambled shRNA.
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