Chromatin profiling following SWI/SNF inhibition in H295R or HEK 293T cells [ChIP-seq]
ChIP-seq / ATAC-seq (genome binding and open chromatin) · human · 25 samples
- What it is
- ChIP-seq / ATAC-seq (genome binding and open chromatin)Where proteins bind the genome, or which regions are open and active.
- Organism
- Homo sapiens (human)
- Samples
- 25 samples: 8 conditions, mostly in duplicateGroups are read from the sample names, not from the paper. Check the list below.
- Data you can get
- A processed table (series matrix) and supplementary files from NCBI; the raw sequencing reads from ENA. Which of these hold counts depends on the study, so check the file list.
- Paper
- β-catenin functions as a molecular adapter for disordered cBAF interactionsMolecular cell · 2025
Direct links to NCBI and ENA, no account and no request form. Pick by what you want to do: take the small processed table to look, the supplementary files to re-analyse, or the original raw data to start over.
Processed table
The series matrix: one table of values per sample as the submitters processed it. The smallest download, and it opens in Excel or R. Start here to see what the data look like.
Supplementary files
The count tables and other files the authors uploaded. Take only the samples you need, or the whole study as GSE260867_RAW.tar (it can run to tens of gigabytes).
Raw sequencing reads
The original FASTQ files, straight from the sequencer, held at ENA. Large, and you align them yourself, so only choose this if you need to re-process the study from scratch.
What the files usually look like: Look for peak files (.bed, .narrowPeak) and signal tracks (.bigWig) among the supplementary files; the raw reads are on ENA.
Also filed as BioProject PRJNA1083884 and SRA study SRP493418. Searching any of these in the dataset finder brings you back here.
Supports a between-group comparison across 16 samples.
How we read this: 8 replicated groups read from 25 sample titles; they account for 16 of them. These groups are guessed from the sample names, not taken from the paper, so check them against the sample list below before relying on them.
- GSM8125845 ChIP_SF1_H295R_DMSO_rep1 ChIP_SF1_H295R_DMSO
- GSM8125849 ChIP_SF1_H295R_DMSO_rep2 ChIP_SF1_H295R_DMSO
- GSM8125852 ChIP_SF1_H295R_BRM014_rep1 ChIP_SF1_H295R_BRM014
- GSM8125856 ChIP_SF1_H295R_BRM014_rep2 ChIP_SF1_H295R_BRM014
- GSM8125859 ChIP_Bcat_H295R_DMSO_rep1 ChIP_Bcat_H295R_DMSO
- GSM8125863 ChIP_Bcat_H295R_DMSO_rep2 ChIP_Bcat_H295R_DMSO
- GSM8125866 ChIP_Bcat_H295R_BRM014_rep1 ChIP_Bcat_H295R_BRM014
- GSM8125869 ChIP_Bcat_H295R_BRM014_rep2 ChIP_Bcat_H295R_BRM014
- GSM8125872 ChIP_ARID1A_H295R_rep1
- GSM8125876 ChIP_PBRM1_H295R_rep1
- GSM8125879 ChIP_BRD9_H295R_rep1
- GSM8125883 ChIP_SMARCA4_H295R_rep1
- GSM8125886 ChIP_CTCF_H295R_rep1
- GSM8125890 ChIP_H3K4me1_H295R_rep1
- GSM8125893 ChIP_H3K4me3_H295R_rep1
- GSM8125897 ChIP_H3K27me3_H295R_rep1
- GSM8125900 ChIP_H3K27ac_H295R_rep1
- GSM8986598 ChIP_p300_293T_DMSO_rep1 ChIP_p300_293T_DMSO
- GSM8986599 ChIP_p300_293T_DMSO_rep2 ChIP_p300_293T_DMSO
- GSM8986600 ChIP_p300_293T_BRM014_rep1 ChIP_p300_293T_BRM014
- GSM8986601 ChIP_p300_293T_BRM014_rep2 ChIP_p300_293T_BRM014
- GSM8986602 ChIP_p300_H295R_DMSO_rep1 ChIP_p300_H295R_DMSO
- GSM8986603 ChIP_p300_H295R_DMSO_rep2 ChIP_p300_H295R_DMSO
- GSM8986604 ChIP_p300_H295R_BRM014_rep1 ChIP_p300_H295R_BRM014
- GSM8986605 ChIP_p300_H295R_BRM014_rep2 ChIP_p300_H295R_BRM014
- GSE256316 SWI/SNF complex-mediated ZNF410 cooperative binding maintains chromatin accessibility and enhancer activity [ChIP-Seq] 121 samples shares swi, snf, chromatin
- GSE261988 SWI/SNF is a gatekeeper of chromatin access at enhancer networks controlling triple-negative breast cancer progression [ChIP-Seq] 79 samples shares swi, snf, chromatin
- GSE215323 Pivotal Role of the NuRD Complex in Remodeling Chromatin Environment and Maintaining Genome Architecture at CTCF Sites [HEK293T_BPK25_ChIP_Seq] 53 samples shares 293t, hek, chromatin
- GSE278192 Pivotal Role of the NuRD Complex in Remodeling Chromatin Environment and Maintaining Genome Architecture at CTCF Sites [HEK293T ChIP-seq] 45 samples shares 293t, hek, chromatin
- GSE253694 Pivotal Role of the NuRD Complex in Remodeling Chromatin Environment and Maintaining Genome Architecture at CTCF Sites [HEK293T_siNURD_CTCF_ChIP_Seq] 36 samples shares 293t, hek, chromatin
- GSE253697 Pivotal Role of the NuRD Complex in Remodeling Chromatin Environment and Maintaining Genome Architecture at CTCF Sites [HEK293T_siNURD_TET1_ChIP_Seq] 36 samples shares 293t, hek, chromatin
- GSE328419 CBFA2T3-GLIS2 fusion-mediated mSWI/SNF chromatin remodeler disruption generates cancer-specific dependencies in AMKL [Cut & Run M-07e] 34 samples shares swi, snf, chromatin
- GSE256317 SWI/SNF complex-mediated ZNF410 cooperative binding maintains chromatin accessibility and enhancer activity [ATAC-Seq] 32 samples shares swi, snf, chromatin
Metadata from NCBI GEO, cached and refreshed periodically — the NCBI page above is authoritative. Downloads link straight to NCBI/ENA; nothing is proxied through BioTransfer.