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TFE3 fusions escape from controlling of mTOR signaling pathway and accumulate in the nucleus promoting genes expression in Xp11.2 translocation renal cell carcinomas.

GSE267212 Homo sapiens Genome binding/occupancy profiling by high throughput sequencing 4 samples Submitted 2024/05/15 Platform GPL11154
Summary
ChIP analysis demonstrated that TFE3 fusions could bind to the promoters of the target genes as a wild-type TFE3 protein.
Published in
TFE3 fusions escape from controlling of mTOR signaling pathway and accumulate in the nucleus promoting genes expression in Xp11.2 translocation renal cell carcinomas
Yin X, Wang B, Gan W et al. · Journal of experimental & clinical cancer research : CR 2019 · PMID 30849994 · doi:10.1186/s13046-019-1101-7
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Also filed as BioProject PRJNA1110249 and SRA study SRP507047. Searching any of these in the dataset finder brings you back here.

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