Metabolic and transcriptomic reprogramming in the transition from a proliferative to a quiescent state is mediated by YAP-dependent and YAP-independent mechanisms.
Direct links to NCBI, no account and no request form: the whole study as GSE272680_RAW.tar, processed values as the series matrix, the supplementary file directory, and per-sample supplementary files for any of the 6 samples. Raw sequencing reads are also available from ENA.
Also filed as BioProject PRJNA1137948 and SRA study SRP521091. Searching any of these in the dataset finder brings you back here.
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+ 6 more — browse all 6 samples with per-sample file links →
- GSE327176 Single-cell transcriptomic profiling unveils tumor-mediated reprogramming of neutrophils and their unique vulnerability that inhibits metastasis 25 samples
- GSE341948 Multi-tissue transcriptomic landscape reveals synergistic mechanisms of exercise and GLP-1 agonist in ameliorating diabetic phenotypes in db/db mice 12 samples
- GSE314817 Multi-tissue, multi-time-point transcriptomic atlas of aging in mice and rats [mouse] 2432 samples
- GSE331176 Phagosome-mediated activation of STING by purine and pyrimidine-based bacterial cyclic dinucleotides 380 samples
- GSE185862 A taxonomy of transcriptomic cell types across the isocortex and hippocampal formation 95 samples
- GSE319035 P21+TREM2+-Senescent Macrophages Fuel Inflammaging and Metabolic Dysfunction-Associated Steatotic Liver Disease [NAFLD_time_course] 77 samples
- GSE319769 Transcriptomic profiling of the intra-amygdala kainate mouse model across the stages of epileptogenesis 36 samples
- GSE235046 Innate immune and metabolic signaling retain damaged mitochondria at cell membranes for mitoxyperilysis 15 samples
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