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Ischemic stroke accelerates atherosclerosis progression via FTO-mediated epigenetic modification

GSE307081 Homo sapiens Expression profiling by high throughput sequencing 7 samples Submitted 2026/07/01 Platform GPL24676
Summary
Residual risk of stroke recurrence persists despite the administration of the prevention measures. The recurrent risk is particularly high in patients with atherosclerosis. However, the underlying mechanisms leading to atherosclerosis progression and recurrent stroke have not been fully understood. We found that stroke accelerated atherosclerosis progression.FTO overexpression suppressed atheroprogression after stroke via inhibiting autophagy-dependent ferroptosis.
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Also filed as BioProject PRJNA1314227 and SRA study SRP616606. Searching any of these in the dataset finder brings you back here.

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