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The FGF4–integrin β1 axis restores directed collective migration to accelerate diabetic wound regeneration.

GSE335568 Homo sapiens Genome binding/occupancy profiling by high throughput sequencing 3 samples Submitted 2026/06/18 Platform GPL34284
Summary
This dataset was generated to investigate the mechanisms underlying the pro-healing effects of Fibroblast Growth Factor 4 (FGF4) on diabetic wounds. We performed RNA sequencing on wound tissues from streptozotocin (STZ)-induced diabetic mice. The mice received daily topical applications of either recombinant FGF4 (rFGF4) or PBS vehicle control. Wound tissues were harvested on day 7 post-wounding for transcriptomic analysis.
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Direct links to NCBI, no account and no request form: the whole study as GSE335568_RAW.tar, processed values as the series matrix, the supplementary file directory, and per-sample supplementary files for any of the 3 samples. Raw sequencing reads are also available from ENA.

Also filed as BioProject PRJNA1478895 and SRA study SRP710103. Searching any of these in the dataset finder brings you back here.

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