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ERα/miR-26a/ANKRD52 axis regulates progression of MASH- associated HCC in HDTVi HCC models

GSE341780 Homo sapiens Expression profiling by high throughput sequencing 6 samples Submitted 2026/07/29 Platform GPL24676
Summary
The lack of robust mouse models for metabolic dysfunction-associated steatohepatitis-hepatocellular carcinoma (MASH-HCC) severely hinders mechanistic investigation and therapeutic drug development. Conventional MASH-HCC animal models require a lengthy induction period of more than 10 months, greatly limiting high-throughput mechanistic studies and drug screening.
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Also filed as BioProject PRJNA1504082 and SRA study SRP722341. Searching any of these in the dataset finder brings you back here.

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