GEO series
Loss of BAP1 cooperates with TP53 deficiency to transform a common myelo-erythroid progenitor (ChIP-seq - mus musculus)
GSE173547
Mus musculus
Genome binding/occupancy profiling by high throughput sequencing
17 samples
2026/03/05
GPL21103
Summary
We performed RNA-seq and ChIP-seq in lineage depleted bone marrow cells isolated from single and double Bap1 and Trp53 knockout mice to identify transcriptional and epigenetic programs that drive erythroleukemia.
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Paper (PMID 40540751) ↗
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