Adenovirus small E1A directs activation of Alu transcription at YAP/TEAD- and AP-1- bound enhancers through interactions with the EP400 chromatin remodeler
ChIP-seq / ATAC-seq (genome binding and open chromatin) · human · 28 samples
- What it is
- ChIP-seq / ATAC-seq (genome binding and open chromatin)Where proteins bind the genome, or which regions are open and active.
- Organism
- Homo sapiens (human)
- Samples
- 28 samples: 28 samples, each with a distinct title — no replicated groupsGroups are read from the sample names, not from the paper. Check the list below.
- Data you can get
- A processed table (series matrix) and supplementary files from NCBI; the raw sequencing reads from ENA. Which of these hold counts depends on the study, so check the file list.
- Paper
- Adenovirus small E1A directs activation of Alu transcription at YAP/TEAD- and AP-1-bound enhancers through interactions with the EP400 chromatin remodelerNucleic acids research · 2024
Direct links to NCBI and ENA, no account and no request form. Pick by what you want to do: take the small processed table to look, the supplementary files to re-analyse, or the original raw data to start over.
Processed table
The series matrix: one table of values per sample as the submitters processed it. The smallest download, and it opens in Excel or R. Start here to see what the data look like.
Supplementary files
The count tables and other files the authors uploaded. Take only the samples you need, or the whole study as GSE208717_RAW.tar (it can run to tens of gigabytes).
Raw sequencing reads
The original FASTQ files, straight from the sequencer, held at ENA. Large, and you align them yourself, so only choose this if you need to re-process the study from scratch.
What the files usually look like: Look for peak files (.bed, .narrowPeak) and signal tracks (.bigWig) among the supplementary files; the raw reads are on ENA.
Also filed as BioProject PRJNA860788 and SRA study SRP387306. Searching any of these in the dataset finder brings you back here.
Supports a case/control comparison: 8 samples read as cases, 5 as controls.
How we read this: Read from 28 sample titles: 28 distinct titles with little repetition. These groups are guessed from the sample names, not taken from the paper, so check them against the sample list below before relying on them.
- GSM6364235 IMR90, dl1500, BDP1, ChIP-seq
- GSM6364236 IMR90, mock, BDP1, ChIP-seq
- GSM6364237 IMR90, dl1500, RPC39, ChIP-seq
- GSM6364238 IMR90, mock, RPC39, ChIP-seq
- GSM6364239 IMR90, dl1500, TFIIIC, ChIP-seq
- GSM6364240 IMR90, mock, TFIIIC, ChIP-seq
- GSM6365504 IMR90, dl1500, replicate 1, RNA-seq
- GSM6365505 IMR90, dl1500, replicate 2, RNA-seq
- GSM6365506 IMR90, mock, replicate 1, RNA-seq
- GSM6365507 IMR90, mock, replicate 2, RNA-seq
- GSM6365508 IMR90, dl312, replicate 1, RNA-seq
- GSM6365509 IMR90, dl312, replicate 2, RNA-seq
- GSM6365510 IMR90, dl1500, replicate 1, RNA-seq mutants
- GSM6365512 IMR90, dl1500, replicate 2, RNA-seq mutants
- GSM6365513 IMR90, mock, replicate 1, RNA-seq mutants
- GSM6365515 IMR90, mock, replicate 2, RNA-seq mutants
- GSM6365516 IMR90, RB-, replicate 1, RNA-seq mutants
- GSM6365517 IMR90, RB-, replicate 2, RNA-seq mutants
- GSM6365518 IMR90, p300-, replicate 1, RNA-seq mutants
- GSM6365520 IMR90, p300-, replicate 2, RNA-seq mutants
- GSM6365521 IMR90, p400-, replicate 1, RNA-seq mutants
- GSM6365523 IMR90, p400-, replicate 2, RNA-seq mutants
- GSM8434430 input for dl312 infection of IMR90
- GSM8434431 ChIP of BDP1 in dl312 infection of IMR90
- GSM8434432 input for dl1500 infection of IMR90
- GSM8434433 ChIP of BDP1 in dl1500 infection of IMR90
- GSM8434434 input for p400b infection of IMR90
- GSM8434435 ChIP of BDP1 in p400b infection of IMR90
- GSE208716 Adenovirus small E1A directs activation of Alu transcription at YAP/TEAD- and AP-1- bound enhancers through interactions with the EP400 chromatin remodeler [RNA-Seq] 16 samples shares ep400, tead, ap-1, e1a, yap, transcription, interactions, adenovirus
- GSE208685 Adenovirus small E1A directs activation of Alu transcription at YAP/TEAD- and AP-1- bound enhancers through interactions with the EP400 chromatin remodeler [ChIP-Seq] 12 samples shares ep400, tead, ap-1, e1a, yap, transcription, interactions, adenovirus
- GSE66083 Widespread association between YAP/TAZ/TEAD and AP-1 at enhancers drives oncogenic growth 25 samples shares tead, ap-1, yap
- GSE66082 Widespread association between YAP/TAZ/TEAD and AP-1 at enhancers drives oncogenic growth [gene expression] 12 samples shares tead, ap-1, yap
- GSE295183 HIF2ɑ Requires Engagement with YAP/TAZ and AP-1 Transcription Factors to Maintain Oncogenic Transcription in Clear Cell Renal Cell Carcinoma [CUT&Tag] 60 samples shares ap-1, yap, transcription
- GSE306447 Aberrant Hippo-YAP/TEAD signaling drives malignant transcriptional reprogramming in external auditory canal squamous cell carcinoma 27 samples shares tead, yap, transcription
- GSE301506 YAP AND TEAD ARE TRANSCRIPTIONAL REGULATORS OF NEUROENDOCRINE DIFFERENTIATION AND GROWTH IN CARCINOID CELLS [ChIP-seq] 20 samples shares tead, yap, transcription
- GSE269900 Identification of resistance mechanisms to small-molecule inhibition of YAP/TEAD-regulated transcription 18 samples shares tead, yap, transcription
Metadata from NCBI GEO, cached and refreshed periodically — the NCBI page above is authoritative. Downloads link straight to NCBI/ENA; nothing is proxied through BioTransfer.