The role of compensatory metabolic reprogramming in ensuring PDAC cell survival under the limitation of glutamine
Direct links to NCBI, no account and no request form: the whole study as GSE229055_RAW.tar, processed values as the series matrix, the supplementary file directory, and per-sample supplementary files for any of the 6 samples. Raw sequencing reads are also available from ENA.
Also filed as BioProject PRJNA952651 and SRA study SRP431141. Searching any of these in the dataset finder brings you back here.
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- GSE327176 Single-cell transcriptomic profiling unveils tumor-mediated reprogramming of neutrophils and their unique vulnerability that inhibits metastasis 25 samples
- GSE235046 Innate immune and metabolic signaling retain damaged mitochondria at cell membranes for mitoxyperilysis 15 samples
- GSE305453 Perturb-seq uncovers pathological obstacles to direct cardiac reprogramming in vivo 15 samples
- GSE324432 A novel sorting method uncovers metabolic heterogeneity between mononucleated and binucleated tetraploid hepatocytes 15 samples
- GSE304391 Global identification of ER stress-regulated RNA binding Proteins in clonal pancreatic beta-cells, reveals an important role for DEAD-box helicase 3 X-linked (DDX3X) in the execution of the Unfolded Protein Response and the determination of cell fate. 12 samples
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Metadata from NCBI GEO, cached and refreshed periodically — the NCBI page above is authoritative. Downloads link straight to NCBI/ENA; nothing is proxied through BioTransfer.