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OLFM4 is a direct target of the cGAS-STING pathway and acts as an extracellular inhibitor for the Hippo signaling in EBV-associated cancer

GSE240275 Homo sapiens Expression profiling by high throughput sequencing 6 samples Submitted 2024/09/18 Platform GPL24676
Summary
Transcriptome analysis of the function of OLFM4 MV on gastric cancer HGC-27 cells
Published in
Epstein-Barr virus infection upregulates extracellular OLFM4 to activate YAP signaling during gastric cancer progression
Wen F, Han Y, Zhang H et al. · Nature communications 2024 · PMID 39627192 · doi:10.1038/s41467-024-54850-6
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Also filed as BioProject PRJNA1003081 and SRA study SRP453756. Searching any of these in the dataset finder brings you back here.

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