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Effect of androgen receptor signaling on H3K27 trimethylation in Th17 cells

GSE241823 Mus musculus Genome binding/occupancy profiling by high throughput sequencing 12 samples 2024/08/27 GPL24247
Summary
Females have increased prevalence of many Th17-mediated diseases, including asthma. Androgen signaling decreases Th17-mediated airway inflammation, and Th17 cells rely on glutaminolysis. However, it remains unclear whether androgen receptor (AR) signaling modifies glutamine metabolism to suppress Th17-mediated inflammation. In this experiment, we conducted a CUT & RUN to determine how AR signaling modified histone 3 lysine 27 trimethylation in Th17 cells from WT males, WT females, and ArTfm male mice (n=3 from each group).
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