Prevalent p53 gain-of-function mutants co-opt epigenetic pathways to drive cancer growth
Direct links to NCBI, no account and no request form: the whole study as GSE59176_RAW.tar, processed values as the series matrix, the supplementary file directory, and per-sample supplementary files for any of the 15 samples. Raw sequencing reads are also available from ENA.
Also filed as BioProject PRJNA254538 and SRA study SRP044136. Searching any of these in the dataset finder brings you back here.
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- GSE254753 Epigenetic memory of radiotherapy in dermal fibroblasts impairs wound repair capacity in cancer survivors (ATAC-Seq) 45 samples
- GSE287736 Sex-specific KDM6A-HNF4A-CREBH network controls lipoprotein cholesterol metabolism and atherosclerosis via epigenetic reprograming of hepatocytes 138 samples
- GSE244947 NOTCH1 reverses immune suppression in small cell lung cancer through reactivation of STING 87 samples
- GSE295336 H2BE113K mutation promotes breast cancer metastasis through modulating chromatin dynamics 77 samples
- GSE292300 A core transcriptional regulatory circuitry controls super-enhancer-driven activation of LGR5 in colorectal cancer: ChIP-seq 76 samples
- GSE242464 Epigenetic regulation of p63 blocks squamous-to-neuroendocrine transdifferentiation in esophageal development and malignancy 53 samples
- GSE274229 Evolution of myeloid-mediated immunotherapy resistance in prostate cancer 52 samples
- GSE291509 A conserved function of EZH2-PRC2 in the repression of primitive endoderm cell fate 43 samples
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