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Disease intelligence · mutation landscape

Breast cancer mutation landscape

How often each gene is altered in breast cancer, in each sequenced cohort, over the patients on whom it could have been called. Copy number is its own row. Nothing is pooled.

Retrieved 2026-09-17 · Reference cohort: brca_tcga_pan_can_atlas_2018 · JSON: /disease/breast-cancer/mutations.json · Back to the briefing

Answer block

In TCGA PanCancer Atlas breast (2018) (1066 sequenced patients, exome or genome), the most frequently altered of the 47 genes shown are PIK3CA 32.46%, TP53 32.46%, GATA3 11.91%, CDH1 11.91%, ERBB2 11.5% (amplification). Each figure divides by the patients on whom that gene could be called.

21 of 1066 patients are hypermutated (more than 370 non-silent mutations, ten times the cohort median of 37); every gene's frequency without them is beside the headline.

Of the briefing's 12 curated targets, 5 are altered in under 2% of this cohort (ESR1, PGR, CDK4, CDK6, TACSTD2): targets by expression, dependency or drug label, not by mutation. Frequency is not targetability, in either direction.

3 cohorts are shown and none are pooled; overlap between them has not been checked and there is no disease-wide percentage.

Evidence boundary: frequency here is a count in a named cohort. Whether an alteration is a driver, is actionable, or has a drug is the briefing's question and is not inferred from these numbers.

What is altered, by cohort

One row per alteration, not per gene: a gene that is amplified and rarely mutated (ERBB2, MYCN, EGFR) gets a row for each. Every cell divides by its own denominator — the patients in that cohort on whom that gene could be called. Copy-number rows are shown only where at least one cohort reaches 2%.

Alterationbrca_tcga_pan_can_atlas_2018
1066 pts · exome or genome
brca_metabric
2433 pts · targeted panel
brca_msk_2025
462 pts · targeted panel
ESR1 SNV / small indel0.84%9/1066·14.07%65/462
ESR1 amplification1.87%20/10702.3%50/21732.16%10/462
PGR SNV / small indel0.66%7/1066·3.5%15/429
ERBB2 SNV / small indel2.81%30/10662.92%71/243310.39%48/462
ERBB2 amplification11.5%123/107015.74%342/217314.5%67/462
PIK3CA SNV / small indel32.46%346/106640.07%975/243359.96%277/462
PIK3CA amplification3.18%34/10704.51%98/21733.25%15/462
AKT1 SNV / small indel2.53%27/10664.03%98/24336.49%30/462
PTEN SNV / small indel5.44%58/10663.9%95/243314.29%66/462
PTEN deep deletion5.14%55/10702.02%44/21731.52%7/462
CDK4 SNV / small indel0%·1.08%5/462
CDK6 SNV / small indel0.28%3/1066·0.87%4/462
BRCA1 SNV / small indel2.53%27/10661.73%42/24338.01%37/462
BRCA2 SNV / small indel2.72%29/10661.85%45/243312.12%56/462
TP53 SNV / small indel32.46%346/106635.39%861/243355.84%258/462
TACSTD2 SNV / small indel0%··
GATA3 SNV / small indel11.91%127/106610.97%267/243313.64%63/462
GATA3 amplification2.99%32/10704.65%101/21732.16%10/462
CDH1 SNV / small indel11.91%127/10669.58%233/243330.74%142/462
KMT2C SNV / small indel9.1%97/106611.39%277/243327.92%129/462
MAP3K1 SNV / small indel8.35%89/10669.7%236/243317.75%82/462
NCOR1 SNV / small indel4.69%50/10663.53%86/243314.07%65/462
MAP2K4 SNV / small indel3.94%42/10663.25%79/24337.79%36/462
MAP2K4 deep deletion2.43%26/10702.12%46/21731.52%7/462
RUNX1 SNV / small indel3.75%40/10663.33%81/24335.41%25/462
RUNX1 amplification1.21%13/10702.39%52/21730.22%1/462
ARID1A SNV / small indel3.66%39/10664.69%114/243317.75%82/462
HUWE1 SNV / small indel3.56%38/1066··
NF1 SNV / small indel3.47%37/10663.74%91/243320.56%95/462
NF1 amplification2.24%24/10701.61%35/21730.43%2/462
CACNA1E SNV / small indel3.47%37/1066··
CACNA1E amplification7.38%79/107020.94%455/21730%
MDN1 SNV / small indel3.38%36/1066··
RELN SNV / small indel3.19%34/1066··
TBX3 SNV / small indel3.1%33/10665.01%122/243313.2%61/462
MYCBP2 SNV / small indel3.1%33/1066··
SPEN SNV / small indel3.0%32/1066·15.8%73/462
KMT2D SNV / small indel3.0%32/10667.23%176/243320.56%95/462
PRUNE2 SNV / small indel2.91%31/1066··
FOXA1 SNV / small indel2.91%31/1066·14.72%68/462
FOXA1 amplification1.5%16/10702.25%49/21731.95%9/462
FAT1 SNV / small indel2.91%31/1066·16.88%78/462
VPS13C SNV / small indel2.81%30/1066··
TEX15 SNV / small indel2.81%30/1066··
TEX15 deep deletion3.64%39/10700.74%16/21730%
MYH9 SNV / small indel2.81%30/10663.58%87/2433·
MXRA5 SNV / small indel2.81%30/1066··
LYST SNV / small indel2.81%30/1066··
LYST amplification9.63%103/107021.54%468/21730%
FCGBP SNV / small indel2.81%30/1066··
FCGBP amplification2.15%23/10701.24%27/21730%
DYNC1H1 SNV / small indel2.81%30/1066··
BLTP1 SNV / small indel2.81%30/1066··
AKAP9 SNV / small indel2.81%30/10665.88%143/2433·
VPS13B SNV / small indel2.72%29/1066··
VPS13B amplification11.03%118/107020.94%455/21730%
UTRN SNV / small indel2.72%29/10665.01%122/2433·
PIK3R1 SNV / small indel2.72%29/10662.47%60/24336.49%30/462
MED12 SNV / small indel2.72%29/1066·6.71%31/462
HERC2 SNV / small indel2.72%29/10666.45%157/2433·
TG SNV / small indel2.63%28/10665.55%135/2433·
TG amplification11.31%121/107023.1%502/21730%

observed — shade scales with frequency, full at 30% assayed, none found not on this cohort's panel cohort not readable

Key findings

PIK3CA is mutated in 346 of 1066 patients in TCGA PanCancer Atlas breast (2018).
Numerator: 346 · Denominator: 1066 · Frequency: 32.46% · Observed in 3 cohorts · Confidence: moderate · Source: brca_tcga_pan_can_atlas_2018 · Retrieved: 2026-09-17

TP53 is mutated in 346 of 1066 patients in TCGA PanCancer Atlas breast (2018).
Numerator: 346 · Denominator: 1066 · Frequency: 32.46% · Observed in 3 cohorts · Confidence: moderate · Source: brca_tcga_pan_can_atlas_2018 · Retrieved: 2026-09-17

GATA3 is mutated in 127 of 1066 patients in TCGA PanCancer Atlas breast (2018).
Numerator: 127 · Denominator: 1066 · Frequency: 11.91% · Observed in 3 cohorts · Confidence: moderate · Source: brca_tcga_pan_can_atlas_2018 · Retrieved: 2026-09-17

Gene table — reference cohort

Headline values are from the reference cohort, brca_tcga_pan_can_atlas_2018; the matrix above keeps every cohort separate. "Curated" marks a gene the disease briefing lists as a target; the rest are here because they are among the most frequently mutated genes in the reference cohort. Recurrent changes are the reference cohort's commonest protein changes.

GeneWhy listedLargest alterationAltered / testedFrequencyWithout hypermutatedCohorts observedRange across cohortsRecurrent changes
ESR1 curated target amplification 20 / 1070 1.87% mutation 0.84% 0.57% 2 / 3 0.84–14.07% E380Q (n=2), A593D (n=1), E523Q (n=1), I451_I452del (n=1), P222S (n=1)
PGR curated target deep deletion 10 / 1070 0.93% mutation 0.66% 0.57% 2 / 3 0.66–3.5% R740Q (n=1), A914V (n=1), E596Gfs*13 (n=1), E833K (n=1), I896M (n=1)
ERBB2 curated target amplification 123 / 1070 11.5% mutation 2.81% 2.3% 3 / 3 2.81–10.39% L755S (n=5), V777L (n=4), D769Y (n=2), I767M (n=2), L755M (n=1)
PIK3CA curated target SNV / small indel 346 / 1066 32.46% 31.96% 3 / 3 32.46–59.96% H1047R (n=129), E545K (n=65), E542K (n=43), N345K (n=18), H1047L (n=13)
AKT1 curated target SNV / small indel 27 / 1066 2.53% 2.58% 3 / 3 2.53–6.49% E17K (n=25), L52R (n=1), V4L (n=1)
PTEN curated target SNV / small indel 58 / 1066 5.44% 4.59% 3 / 3 3.9–14.29% R130Q (n=4), R130* (n=2), T319* (n=2), K267Rfs*9 (n=2), N48Tfs*6 (n=2)
CDK4 curated target amplification 14 / 1070 1.31% mutation 0.0% 0.0% 1 / 3 0.0–1.08% none recurrent
CDK6 curated target amplification 6 / 1070 0.56% mutation 0.28% 0.19% 2 / 3 0.28–0.87% T88A (n=1), H139Q (n=1), P118S (n=1)
BRCA1 curated target SNV / small indel 27 / 1066 2.53% 2.2% 3 / 3 1.73–8.01% Q934* (n=1), E9Q (n=1), X27_splice (n=1), E720* (n=1), T688Vfs*12 (n=1)
BRCA2 curated target SNV / small indel 29 / 1066 2.72% 2.3% 3 / 3 1.85–12.12% Q3206E (n=1), Y3308* (n=1), L2926* (n=1), D1355Y (n=1), E260Sfs*15 (n=1)
TP53 curated target SNV / small indel 346 / 1066 32.46% 32.54% 3 / 3 32.46–55.84% R175H (n=21), R273H (n=13), R196* (n=8), Y220C (n=7), R342* (n=7)
TACSTD2 curated target amplification 16 / 1070 1.5% mutation 0.0% 0.0% 0 / 3 0.0–0.0% none recurrent
GATA3 by frequency SNV / small indel 127 / 1066 11.91% 11.67% 3 / 3 10.97–13.64% X308_splice (n=21), P408Afs*99 (n=13), D335Gfs*17 (n=8), M293K (n=3), S436Lfs*71 (n=2)
CDH1 by frequency SNV / small indel 127 / 1066 11.91% 11.58% 3 / 3 9.58–30.74% Q23* (n=8), R63* (n=4), P127Afs*41 (n=4), E243K (n=3), X722_splice (n=3)
KMT2C by frequency SNV / small indel 97 / 1066 9.1% 8.23% 3 / 3 9.1–27.92% R4478* (n=2), E640* (n=2), Q1478* (n=2), S1334C (n=1), E2838K (n=1)
MAP3K1 by frequency SNV / small indel 89 / 1066 8.35% 8.23% 3 / 3 8.35–17.75% R273Sfs*27 (n=3), R763Cfs*35 (n=3), V1346del (n=2), L319Tfs*7 (n=2), Q1048* (n=2)
NCOR1 by frequency SNV / small indel 50 / 1066 4.69% 4.21% 3 / 3 3.53–14.07% T525Nfs*11 (n=2), P7S (n=1), E531Q (n=1), G1163D (n=1), E592* (n=1)
MAP2K4 by frequency SNV / small indel 42 / 1066 3.94% 4.02% 3 / 3 3.25–7.79% R281* (n=2), S251I (n=2), X75_splice (n=2), D263Cfs*4 (n=2), S184L (n=2)
RUNX1 by frequency SNV / small indel 40 / 1066 3.75% 3.54% 3 / 3 3.33–5.41% D123Gfs*15 (n=5), R169Kfs*44 (n=2), D123Gfs*11 (n=2), D123Mfs*10 (n=1), A244V (n=1)
ARID1A by frequency SNV / small indel 39 / 1066 3.66% 3.35% 3 / 3 3.66–17.75% X960_splice (n=2), R1989* (n=2), Q528* (n=2), Q944* (n=1), R2143C (n=1)
HUWE1 by frequency SNV / small indel 38 / 1066 3.56% 2.49% 1 / 3 3.56–3.56% S3122F (n=1), A889T (n=1), R3174H (n=1), D882N (n=1), R3786Q (n=1)
NF1 by frequency SNV / small indel 37 / 1066 3.47% 3.06% 3 / 3 3.47–20.56% Q209* (n=1), W571* (n=1), R366* (n=1), E524Q (n=1), R2119Qfs*10 (n=1)
CACNA1E by frequency amplification 79 / 1070 7.38% mutation 3.47% 2.87% 1 / 3 3.47–3.47% R384C (n=2), R1396C (n=1), G2125V (n=1), Q110K (n=1), X391_splice (n=1)
MDN1 by frequency SNV / small indel 36 / 1066 3.38% 2.58% 1 / 3 3.38–3.38% L423F (n=1), K4830N (n=1), E2201Q (n=1), D2612H (n=1), D5539G (n=1)
RELN by frequency SNV / small indel 34 / 1066 3.19% 2.49% 1 / 3 3.19–3.19% G3444E (n=1), R1385I (n=1), F1269C (n=1), G2175S (n=1), D1175H (n=1)
TBX3 by frequency SNV / small indel 33 / 1066 3.1% 2.87% 3 / 3 3.1–13.2% H187Y (n=2), Y265Lfs*12 (n=1), P134S (n=1), M184Gfs*25 (n=1), K175* (n=1)
MYCBP2 by frequency SNV / small indel 33 / 1066 3.1% 2.3% 1 / 3 3.1–3.1% K2740M (n=1), I3077V (n=1), Q3845* (n=1), S143* (n=1), H3616Y (n=1)
SPEN by frequency SNV / small indel 32 / 1066 3.0% 2.2% 2 / 3 3.0–15.8% N1429K (n=1), P2059Kfs*3 (n=1), F609Lfs*2 (n=1), E2846K (n=1), E2201Q (n=1)
KMT2D by frequency SNV / small indel 32 / 1066 3.0% 2.11% 3 / 3 3.0–20.56% K1686Nfs*36 (n=2), T1681N (n=1), P3246Tfs*5 (n=1), E3587V (n=1), A2119Rfs*36 (n=1)
PRUNE2 by frequency SNV / small indel 31 / 1066 2.91% 2.11% 1 / 3 2.91–2.91% S576N (n=1), H2092Y (n=1), D830V (n=1), V2775M (n=1), S2432F (n=1)
FOXA1 by frequency SNV / small indel 31 / 1066 2.91% 2.68% 2 / 3 2.91–14.72% S250F (n=3), I176M (n=3), D226G (n=2), S194Afs*127 (n=1), I450M (n=1)
FAT1 by frequency SNV / small indel 31 / 1066 2.91% 2.11% 2 / 3 2.91–16.88% S2060F (n=2), T2261M (n=1), D2373N (n=1), K1044R (n=1), D2076N (n=1)
VPS13C by frequency SNV / small indel 30 / 1066 2.81% 1.91% 1 / 3 2.81–2.81% T3732M (n=2), V3571I (n=1), E1451K (n=1), A840D (n=1), A1513S (n=1)
TEX15 by frequency deep deletion 39 / 1070 3.64% mutation 2.81% 2.49% 1 / 3 2.81–2.81% L2764F (n=1), S1432T (n=1), P1734L (n=1), T208K (n=1), E57Q (n=1)
MYH9 by frequency SNV / small indel 30 / 1066 2.81% 2.3% 2 / 3 2.81–3.58% E530K (n=2), P726S (n=1), E1011Q (n=1), A1659T (n=1), L92V (n=1)
MXRA5 by frequency SNV / small indel 30 / 1066 2.81% 2.11% 1 / 3 2.81–2.81% G798S (n=1), G473S (n=1), M273I (n=1), A1492G (n=1), E266* (n=1)
LYST by frequency amplification 103 / 1070 9.63% mutation 2.81% 2.01% 1 / 3 2.81–2.81% Q3352P (n=1), Q2237* (n=1), E1095K (n=1), V1744F (n=1), C2828* (n=1)
FCGBP by frequency SNV / small indel 30 / 1066 2.81% 2.2% 1 / 3 2.81–2.81% N4225Tfs*173 (n=2), L1156R (n=1), G4218S (n=1), V3268I (n=1), E2651K (n=1)
DYNC1H1 by frequency SNV / small indel 30 / 1066 2.81% 1.82% 1 / 3 2.81–2.81% K4418N (n=1), E1786K (n=1), V3797L (n=1), E4439G (n=1), V88I (n=1)
BLTP1 by frequency SNV / small indel 30 / 1066 2.81% 2.39% 1 / 3 2.81–2.81% R3026T (n=1), R2025K (n=1), R3280Kfs*10 (n=1), E299* (n=1), E2425A (n=1)
AKAP9 by frequency SNV / small indel 30 / 1066 2.81% 2.3% 2 / 3 2.81–5.88% G3802* (n=1), E756Kfs*9 (n=1), T1942I (n=1), S3576Y (n=1), E3074Q (n=1)
VPS13B by frequency amplification 118 / 1070 11.03% mutation 2.72% 2.11% 1 / 3 2.72–2.72% G567E (n=1), I737T (n=1), V840M (n=1), P721T (n=1), G2706V (n=1)
UTRN by frequency SNV / small indel 29 / 1066 2.72% 1.91% 2 / 3 2.72–5.01% R2739T (n=1), D808H (n=1), E2888K (n=1), Q750E (n=1), E1688Q (n=1)
PIK3R1 by frequency SNV / small indel 29 / 1066 2.72% 2.39% 3 / 3 2.47–6.49% K567_L570del (n=3), K575_T576dup (n=1), E468_E469insGLYE (n=1), K459_S460dup (n=1), L380del (n=1)
MED12 by frequency SNV / small indel 29 / 1066 2.72% 2.3% 2 / 3 2.72–6.71% Q2159R (n=1), Q323* (n=1), F833L (n=1), K174N (n=1), D234V (n=1)
HERC2 by frequency SNV / small indel 29 / 1066 2.72% 1.91% 2 / 3 2.72–6.45% R1744* (n=1), L294_L296del (n=1), I1210M (n=1), G3189E (n=1), G279V (n=1)
TG by frequency amplification 121 / 1070 11.31% mutation 2.63% 2.11% 2 / 3 2.63–5.55% S1139L (n=1), W1554C (n=1), D1628Y (n=1), G1045R (n=1), Q1741H (n=1)

Cohorts

Listed in the disease profile, not searched: a name search returns the same patients under several accessions. Patients are unique patient ids in the study's sequenced sample list. Hypermutated: more than ten times the cohort's median non-silent mutations per sample, and at least 100.

CohortAccessionPatientsSamples sequenced / in studyAssayPanels (samples)BuildProfiles readHypermutated patientsMedian mutations / sample
TCGA PanCancer Atlas breast (2018) reference
Breast Invasive Carcinoma (TCGA, PanCancer Atlas)
brca_tcga_pan_can_atlas_20181066 observed1066 / 1084exome or genomeWES (1066)hg19SNV, small indel, amplification, deep deletion, structural variant (profile present, not read)2137.0
METABRIC (Nature 2012, Nat Commun 2016)
Breast Cancer (METABRIC, Nature 2012 & Nat Commun 2016)
brca_metabric2433 observed2433 / 2509targeted panelMETABRIC_173 (2433)hg19SNV, small indel, amplification, deep deletion05
MSK breast carcinoma (2025)
Breast Carcinoma (MSK, NPJ Precis Oncol 2025)
brca_msk_2025462 observed527 / 527targeted panelIMPACT468 (283), IMPACT505 (124), IMPACT410 (81), IMPACT341 (39)hg19SNV, small indel, amplification, deep deletion, structural variant (profile present, not read)216

Copy-number events

Discrete calls from each study's copy-number profile: 2 is high-level amplification, −2 deep deletion. Gains and shallow losses are not counted. Denominators are the cohort's copy-number sample list, which differs from its sequenced list. Rows at 2% or more.

GeneEventObserved patientsTested patientsFrequencyCohortProfile
TGamplification502217323.1%brca_metabricbrca_metabric_cna
LYSTamplification468217321.54%brca_metabricbrca_metabric_cna
CACNA1Eamplification455217320.94%brca_metabricbrca_metabric_cna
VPS13Bamplification455217320.94%brca_metabricbrca_metabric_cna
ERBB2amplification342217315.74%brca_metabricbrca_metabric_cna
ERBB2amplification6746214.5%brca_msk_2025brca_msk_2025_gistic
ERBB2amplification123107011.5%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
TGamplification121107011.31%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
VPS13Bamplification118107011.03%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
LYSTamplification10310709.63%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
CACNA1Eamplification7910707.38%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
PTENdeep deletion5510705.14%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
GATA3amplification10121734.65%brca_metabricbrca_metabric_cna
PIK3CAamplification9821734.51%brca_metabricbrca_metabric_cna
TEX15deep deletion3910703.64%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
PIK3CAamplification154623.25%brca_msk_2025brca_msk_2025_gistic
PIK3CAamplification3410703.18%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
GATA3amplification3210702.99%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
MAP2K4deep deletion2610702.43%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
RUNX1amplification5221732.39%brca_metabricbrca_metabric_cna
ESR1amplification5021732.3%brca_metabricbrca_metabric_cna
FOXA1amplification4921732.25%brca_metabricbrca_metabric_cna
NF1amplification2410702.24%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
ESR1amplification104622.16%brca_msk_2025brca_msk_2025_gistic
GATA3amplification104622.16%brca_msk_2025brca_msk_2025_gistic
FCGBPamplification2310702.15%brca_tcga_pan_can_atlas_2018brca_tcga_pan_can_atlas_2018_gistic
MAP2K4deep deletion4621732.12%brca_metabricbrca_metabric_cna
PTENdeep deletion4421732.02%brca_metabricbrca_metabric_cna

Cohort-aware frequencies

Each row is calculated from unique patients in that study's sequenced sample list. The range is descriptive; no pooled estimate is shown because cross-study overlap and assay comparability have not been checked.

GeneRangePer cohort (altered / tested)
ESR10.84–14.07%brca_tcga_pan_can_atlas_2018: 9/1066 (0.84%) · brca_metabric: not assayed · brca_msk_2025: 65/462 (14.07%)
PGR0.66–3.5%brca_tcga_pan_can_atlas_2018: 7/1066 (0.66%) · brca_metabric: not assayed · brca_msk_2025: 15/429 (3.5%)
ERBB22.81–10.39%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: 71/2433 (2.92%) · brca_msk_2025: 48/462 (10.39%)
PIK3CA32.46–59.96%brca_tcga_pan_can_atlas_2018: 346/1066 (32.46%) · brca_metabric: 975/2433 (40.07%) · brca_msk_2025: 277/462 (59.96%)
AKT12.53–6.49%brca_tcga_pan_can_atlas_2018: 27/1066 (2.53%) · brca_metabric: 98/2433 (4.03%) · brca_msk_2025: 30/462 (6.49%)
PTEN3.9–14.29%brca_tcga_pan_can_atlas_2018: 58/1066 (5.44%) · brca_metabric: 95/2433 (3.9%) · brca_msk_2025: 66/462 (14.29%)
CDK40.0–1.08%brca_tcga_pan_can_atlas_2018: 0/1066 (0.0%) · brca_metabric: not assayed · brca_msk_2025: 5/462 (1.08%)
CDK60.28–0.87%brca_tcga_pan_can_atlas_2018: 3/1066 (0.28%) · brca_metabric: not assayed · brca_msk_2025: 4/462 (0.87%)
BRCA11.73–8.01%brca_tcga_pan_can_atlas_2018: 27/1066 (2.53%) · brca_metabric: 42/2433 (1.73%) · brca_msk_2025: 37/462 (8.01%)
BRCA21.85–12.12%brca_tcga_pan_can_atlas_2018: 29/1066 (2.72%) · brca_metabric: 45/2433 (1.85%) · brca_msk_2025: 56/462 (12.12%)
TP5332.46–55.84%brca_tcga_pan_can_atlas_2018: 346/1066 (32.46%) · brca_metabric: 861/2433 (35.39%) · brca_msk_2025: 258/462 (55.84%)
TACSTD20.0–0.0%brca_tcga_pan_can_atlas_2018: 0/1066 (0.0%) · brca_metabric: not assayed · brca_msk_2025: not assayed
GATA310.97–13.64%brca_tcga_pan_can_atlas_2018: 127/1066 (11.91%) · brca_metabric: 267/2433 (10.97%) · brca_msk_2025: 63/462 (13.64%)
CDH19.58–30.74%brca_tcga_pan_can_atlas_2018: 127/1066 (11.91%) · brca_metabric: 233/2433 (9.58%) · brca_msk_2025: 142/462 (30.74%)
KMT2C9.1–27.92%brca_tcga_pan_can_atlas_2018: 97/1066 (9.1%) · brca_metabric: 277/2433 (11.39%) · brca_msk_2025: 129/462 (27.92%)
MAP3K18.35–17.75%brca_tcga_pan_can_atlas_2018: 89/1066 (8.35%) · brca_metabric: 236/2433 (9.7%) · brca_msk_2025: 82/462 (17.75%)
NCOR13.53–14.07%brca_tcga_pan_can_atlas_2018: 50/1066 (4.69%) · brca_metabric: 86/2433 (3.53%) · brca_msk_2025: 65/462 (14.07%)
MAP2K43.25–7.79%brca_tcga_pan_can_atlas_2018: 42/1066 (3.94%) · brca_metabric: 79/2433 (3.25%) · brca_msk_2025: 36/462 (7.79%)
RUNX13.33–5.41%brca_tcga_pan_can_atlas_2018: 40/1066 (3.75%) · brca_metabric: 81/2433 (3.33%) · brca_msk_2025: 25/462 (5.41%)
ARID1A3.66–17.75%brca_tcga_pan_can_atlas_2018: 39/1066 (3.66%) · brca_metabric: 114/2433 (4.69%) · brca_msk_2025: 82/462 (17.75%)
HUWE13.56–3.56%brca_tcga_pan_can_atlas_2018: 38/1066 (3.56%) · brca_metabric: not assayed · brca_msk_2025: not assayed
NF13.47–20.56%brca_tcga_pan_can_atlas_2018: 37/1066 (3.47%) · brca_metabric: 91/2433 (3.74%) · brca_msk_2025: 95/462 (20.56%)
CACNA1E3.47–3.47%brca_tcga_pan_can_atlas_2018: 37/1066 (3.47%) · brca_metabric: not assayed · brca_msk_2025: not assayed
MDN13.38–3.38%brca_tcga_pan_can_atlas_2018: 36/1066 (3.38%) · brca_metabric: not assayed · brca_msk_2025: not assayed
RELN3.19–3.19%brca_tcga_pan_can_atlas_2018: 34/1066 (3.19%) · brca_metabric: not assayed · brca_msk_2025: not assayed
TBX33.1–13.2%brca_tcga_pan_can_atlas_2018: 33/1066 (3.1%) · brca_metabric: 122/2433 (5.01%) · brca_msk_2025: 61/462 (13.2%)
MYCBP23.1–3.1%brca_tcga_pan_can_atlas_2018: 33/1066 (3.1%) · brca_metabric: not assayed · brca_msk_2025: not assayed
SPEN3.0–15.8%brca_tcga_pan_can_atlas_2018: 32/1066 (3.0%) · brca_metabric: not assayed · brca_msk_2025: 73/462 (15.8%)
KMT2D3.0–20.56%brca_tcga_pan_can_atlas_2018: 32/1066 (3.0%) · brca_metabric: 176/2433 (7.23%) · brca_msk_2025: 95/462 (20.56%)
PRUNE22.91–2.91%brca_tcga_pan_can_atlas_2018: 31/1066 (2.91%) · brca_metabric: not assayed · brca_msk_2025: not assayed
FOXA12.91–14.72%brca_tcga_pan_can_atlas_2018: 31/1066 (2.91%) · brca_metabric: not assayed · brca_msk_2025: 68/462 (14.72%)
FAT12.91–16.88%brca_tcga_pan_can_atlas_2018: 31/1066 (2.91%) · brca_metabric: not assayed · brca_msk_2025: 78/462 (16.88%)
VPS13C2.81–2.81%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: not assayed · brca_msk_2025: not assayed
TEX152.81–2.81%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: not assayed · brca_msk_2025: not assayed
MYH92.81–3.58%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: 87/2433 (3.58%) · brca_msk_2025: not assayed
MXRA52.81–2.81%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: not assayed · brca_msk_2025: not assayed
LYST2.81–2.81%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: not assayed · brca_msk_2025: not assayed
FCGBP2.81–2.81%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: not assayed · brca_msk_2025: not assayed
DYNC1H12.81–2.81%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: not assayed · brca_msk_2025: not assayed
BLTP12.81–2.81%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: not assayed · brca_msk_2025: not assayed
AKAP92.81–5.88%brca_tcga_pan_can_atlas_2018: 30/1066 (2.81%) · brca_metabric: 143/2433 (5.88%) · brca_msk_2025: not assayed
VPS13B2.72–2.72%brca_tcga_pan_can_atlas_2018: 29/1066 (2.72%) · brca_metabric: not assayed · brca_msk_2025: not assayed
UTRN2.72–5.01%brca_tcga_pan_can_atlas_2018: 29/1066 (2.72%) · brca_metabric: 122/2433 (5.01%) · brca_msk_2025: not assayed
PIK3R12.47–6.49%brca_tcga_pan_can_atlas_2018: 29/1066 (2.72%) · brca_metabric: 60/2433 (2.47%) · brca_msk_2025: 30/462 (6.49%)
MED122.72–6.71%brca_tcga_pan_can_atlas_2018: 29/1066 (2.72%) · brca_metabric: not assayed · brca_msk_2025: 31/462 (6.71%)
HERC22.72–6.45%brca_tcga_pan_can_atlas_2018: 29/1066 (2.72%) · brca_metabric: 157/2433 (6.45%) · brca_msk_2025: not assayed
TG2.63–5.55%brca_tcga_pan_can_atlas_2018: 28/1066 (2.63%) · brca_metabric: 135/2433 (5.55%) · brca_msk_2025: not assayed

What this page does not do

Structural variants
Read the structural-variant profiles the studies carry; fusions are the defining event in several of these diseases.

Context
Stage, subtype, age and treatment line are not attached to any count; the cohorts differ on all four.

Interpretation
Activating versus inactivating, actionable versus not, and evidence level are not inferred here; the briefing's target table carries the drug and trial facts.

Limitations

How a machine should read this page

  1. Denominators: every frequency divides by the patients in one named cohort on whom the gene could be called; there is no disease-wide figure.
  2. Missing values: not_assayed (the panel did not carry the gene), not_observed (assayed, none found) and not_evaluable (the cohort could not be read) are three different facts and are never converted to zero.
  3. Counting: patients, not samples; several samples from one patient count once. Non-silent calls only.
  4. Copy number: a separate assay with a separate roster; discrete calls at ±2 only.
  5. Hypermutation: flagged per cohort; the headline keeps all patients and the frequency without them is reported beside it.
  6. Provenance: every value carries the study id, the retrieval date and the processing version; the source is the cBioPortal public API.

Machine endpoints: full landscape · genes · cohorts · the disease's own facts: /disease/breast-cancer.json.

Built by the BioTransfer briefings pipeline from the cBioPortal public API. The neuroblastoma page was assembled by hand and set the rules this page follows; how these are built.