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Identify WT and p53 3KR target in basal progenitor cells of epidermis

GSE275726 Mus musculus Genome binding/occupancy profiling by high throughput sequencing 6 samples Submitted 2024/08/31 Platform GPL17021
Summary
Unbalanced and growth promoting cell fate dynamics that follow p53 loss, and the inability of canonical p53 functions to restrict clonal expansion, suggest the existence of a non-canonical p53 transcriptional program that controls key cell fate regulators. We hypothesized that these cell fate genes are among the shared targets of p53WT and p533KR.
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Also filed as BioProject PRJNA1152794 and SRA study SRP528809. Searching any of these in the dataset finder brings you back here.

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