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Regulation of GLIS3 in neonatal kidney development

GSE240532 Mus musculus Expression profiling by high throughput sequencing 24 samples 2024/12/18 GPL19057GPL24247
Summary
Loss of transcription factor GLIS3 function in humans and mice leads to the development of neonatal polycystic kidney disease (PKD). To investigate how loss of GLIS3 function in kidney affects postnatal kidney development and PKD, we analyzed the gene expression profiles of kidneys from WT and Glis3-null mice at P7, P14, and P28 by RNA-Seq analysis using Glis3 global KO model Glis3-mCherry.
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NCBI GEO page ↗ Paper (PMID 39505148) ↗ {# Names what the click gives you. "Open in finder" meant nothing to a visitor who arrived from a search engine and has never seen the tool. #} Find more mouse RNA-seq datasets →
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