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Gene expression data from acetaminophen-induced toxicity in wild-type and Gα12 knockout mice

GSE173595 Mus musculus Expression profiling by high throughput sequencing 12 samples 2024/04/21 GPL17021
Summary
Gα12 is an alpha subunit of heterotrimeric G proteins that activates the downstream pathways from G protein-coupled receptor (GPCR) signals. Gα12 overexpression enhances hepatocellular carcinoma (HCC) malignancy, promoting cancer migration and microvascular invasion. Also, it activates hepatic stellate cells (HSCs) and thereby facilitates the progression of liver fibrosis. However, upregulated Gα12 in hepatocytes by acetaminophen (APAP) mechanically has been scarcely explored. This study provides an evaluation of changes in gene expression using liver samples obtained from WT and Gα12KO mice treated with APAP.
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NCBI GEO page ↗ Paper (PMID 35198058) ↗ {# Names what the click gives you. "Open in finder" meant nothing to a visitor who arrived from a search engine and has never seen the tool. #} Find more mouse RNA-seq datasets →
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